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1. Clinically there was no evidence of portal hypertension.

2. Conversely, there was no significant association with portal hypertension.

3. Our patient thus failed to show evidence of portal hypertension.

4. Discussion Portal hypertension usually complicates the evolution of chronic liver diseases.

5. However spontaneous portal vein thrombus may occur in portal hypertension and the infusion of vasopressin may also play a part.

6. Portal hypertension with stasis and pooling of blood in the reticuloendothelial meshwork may interfere with this process.

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7. Materials and Methods: 90 patients with portal hypertension, and 86 normal subjects had their blood flow parameters in inferior epigastric artery and veins assessed by 2D and CDFI ultrasound.

8. The features cirrhotic portal hypertension were within liver, larger in spleen and widen in portal vein.

9. One of the most common findings with portal hypertension is splenomegaly as seen here.

10. Purpose:To study the value of DSA in treating portal hypertension by subtotal splenectomy with retroperitoneal splenic trans position and devascularization.

11. Chronic constipation, chronic diarrhea, pregnancy, and portal hypertension enhance hemorrhoid formation.

12. Patients described in published works with portopulmonary hypertension have all had clinically obvious portal hypertension.

13. Over half of the patients exhibit clinical signs associated with portal hypertension, such as ascites and hepatorenal syndrome.

14. Decisions regarding appropriate organ transplantation may depend on whether pulmonary hypertension is primary or secondary to portal hypertension.

15. Complete or partial occlusion of the portal vein is a common cause of portal hypertension in childhood.

16. Recently Hadengue etal reported a higher prevalence of 2% in 507 patients with portal hypertension.

17. Many treatments have evolved over the last 30 years for controlling upper gastrointestinal haemorrhage in patients with cirrhosis and portal hypertension.

18. Gastric varices Gastric varices are not uncommon in cirrhotic patients with portal hypertension.

19. Treatment was discontinued when patients developed evidence of cirrhosis or portal hypertension.

20. As mentioned previously, all cases of portopulmonary hypertension reported have had obvious portal hypertension.

21. Pericardial devascularization plus endoscopic variceal ligation(EVL) was applied in our hospital as a new union devascularization therapy for 66 cirrhotic portal hypertension patients since 1993.

22. Objective To study the hemodynamic effects of thalidomide on experimental portal hypertension rats and its relation with tumor necrosis factor (TNF).

23. Objective : To investigate the clinical significance of paraumbilical vein ( PUV ) patefaction extent of portal hypertension.

24. Objective To study the cause, treatment and prevention of gastric rupture after pericardial devascularization plus splenectomy in the treatment of portal hypertension.

25. Prof. Sarin: Bleeding from gastric varices is a major cause of death in portal hypertension.

26. The authors studied the pre and post-operative portography via gastroepiploic vein catheterization performed in 8 patients with portal hypertension of various degrees.

27. Methods Liceratures of overseas main studies in hyperdynamic circulatory syndrome of portal hypertension in recent 10 years were reviewed.

28. Objective:To evaluate the clinical value of spiral CT during arterial portography (SCTAP) in judgment of cirrhosis and portal hypertension.

29. The limited side-to-side portal-caval shunt with a limitary ring attached is the first-choice surgical procedure to treat the portal hypertension in our department.

30. Objective: To probe the effect of application of artificial blood vessel with ring in port acaval shunt for portal hypertension.